A small study (n=38) found no clear link between neurological Long COVID and a shared autoantibody in cerebrospinal fluid.
Immunostaining patterns in brain tissue were similar between patients and controls, and no single autoantigen stood out.
https://www.croiconference.org/abstract/1252-2025/
Immunostaining patterns in brain tissue were similar between patients and controls, and no single autoantigen stood out.
https://www.croiconference.org/abstract/1252-2025/
Comments
How this translate with previous study with aabs in mice? Why every time there is always a promising study for us it seems to be contradicted by another 6 months later? 😵💫
Would have been interesting (as usual) to include PEM as criteria to define LC + larger sample? 📌
Thanks for posting as always, really really grateful 🙏
Thanks again for answering and all the info.